The role of chemical transport in the brown-rot decay resistance of modified woodShow others and affiliations
2016 (English)In: International Wood Products Journal, ISSN 2042-6445, E-ISSN 2042-6453, Vol. 7, no 2, p. 66-70Article in journal (Refereed) Published
Abstract [en]
Chemical modification of wood increases decay resistance but the exact mechanisms remain poorly understood. Recently, Ringman and coauthors examined established theories addressing why modified wood has increased decay resistance and concluded that the most probable cause of inhibition and/or delay of initiation of brown-rot decay is lowering the equilibrium moisture content. In another recent study, Jakes and coauthors examined moisture-induced wood damage mechanisms, including decay and fastener corrosion, and observed that these mechanisms require chemical transport through wood cell walls. They proposed that chemical transport within wood cell walls is controlled by a moisture-induced glass transition in interconnected networks of hemicelluloses and amorphous cellulose. This paper shows how these models jointly suggest mechanisms by which wood modifications can inhibit brown-rot. Alternative mechanisms are also discussed. These models can be used to understand and further improve the performance of wood modification systems. © 2016 IWSc, The Wood Technology Society of the Institute of Materials, Minerals and Mining.
Place, publisher, year, edition, pages
Taylor and Francis Ltd. , 2016. Vol. 7, no 2, p. 66-70
Keywords [en]
Brown-rot decay, Chemical transport, Diffusion, Glass transition temperature, Percolation theory, Wood modification, Cellulose, Chemical modification, Chemical resistance, Glass, Glass transition, Moisture, Moisture control, Solvents, Wood, Amorphous cellulose, Chemical modification of wood, Equilibrium moisture contents, Interconnected network, Decay (organic), Brown Rot Fungi, Corrosion, Decay Resistance, Hemicelluloses
National Category
Engineering and Technology
Identifiers
URN: urn:nbn:se:ri:diva-43913DOI: 10.1080/20426445.2016.1161867Scopus ID: 2-s2.0-84981283742OAI: oai:DiVA.org:ri-43913DiVA, id: diva2:1393048
2020-02-142020-02-142020-12-01Bibliographically approved